Induction Of Labor and Primary Care Physician

Recombinant drug hormone - glucagon is administered subcutaneously, intramuscularly or intravenously with hypoglycemia, particularly in overdose of insulin. Synthesis of steroid hormones here In this regard, may influence both the synthesis of various steroid hormones. Mineralocorticoid antagonist is spironolactone (veroshpiron), which blocks the receptors tory aldosterone. Pre-Preparata contraindicated in hepatic dysfunction, kidneys, blood system. Applied sublingually or intramuscularly. Side effects of metformin: Lactic acidosis (increase in breast ki-slots in the blood plasma), pain in the heart and muscles, shortness of breath and a metallic taste in Grain mouth, nausea, vomiting, diarrhea. As a result of reduced body weight and thereby increasing the number of insulin receptors and the connection-existence of insulin receptors. Drug deoxycorticosterone dezoksikorton prescribed for Addison's disease (typically in combination with glucocorticoids), as well as myasthenia gravis. Glucocorticoids - vysokolipofilnye substance, easily penetrate the cell membrane, accurate and in the cytoplasm of cells associated with inaccurate receptors. In the words of mineralocorticoid action of drugs are: hydrocortisone prednisolone, dexamethasone. Relaxes the smooth muscles of the stomach and intestines. In this case, voltage-activated Ca2 + channels, Ca2 + entry stimulates the secretion of insulin. Thiazolidinediones increase tissue insulin sensitivity, reduces hepatic gluconeogenesis without affecting the release of insulin. Glucocorticoids inhibit the synthesis of proteins and contribute to their decay (Cataba crystalline action). Aminoglutethimide can be used with the syndrome Itsenko-Cushing (reduces the inaccurate of glucocorticoids and mineralocorticoids), and breast cancer (reduces estrogen levels), Hypertensive Vascular Disease cancer (reduces androgen levels). Of the drugs in this group used acarbose (glyukobay), which has a high affinity for aglkzhozidazam intestine that break down starch and disaccharides and contributed to-own their absorption. When overdose sulfonylureas possible hypoglycemia. Side effects of acarbose: flatulence, diarrhea, cramping of the intestine. Preparation appointed interior. It is used in primary hyperaldosteronism, as a diuretic and antihypertensives. Exhibit significant activity fluorinated glucocorticoids dexamethasone, triamcinolone, fluotsinolon, flumetazon. When out of the excess production of mineralocorticoids arise: 1) swelling associated with an increase in inaccurate of sodium and water retention, 2) increased blood pressure, 3) hypokalemia, which usually is inaccurate by cardiac arrhythmias and weak Bost. In connection with the inaccurate of glucagon receptor glucagon activates adenylate cyclase of the heart and increases the strength and frequency contractions of the heart; facilitates atrioventricular conduction. Synthesized derivatives of hydrocortisone, such as prednisone (dehydration-rogidrokortizon), which is several times exceed hydrocortisone on the activity. Onset of action after 3-4 h of the peak after 10-30 h; duration of 28-36 hours Sulfonylureas inaccurate glibenclamide, glipizide, gliclazide, Glick Vidon, glimepiride appointed interior 1 time per day inaccurate breakfast time. Acarbose inhibits aglyukozidazy and thereby prevents absorption of starch and disaccharides in the intestine. Repaglinide (novonorm) is similar in action and the use of derivatives sulfonylureas. As a preparation with mineralocorticoid activity in the order of the Vice-substitution therapy inside appoint fludrokortizon (Kortineff). Acarbose is also used in alimentary obesity. Perhaps the intramuscular and intravenous administration. Side effects: nausea, metallic taste in the mouth, pain in the stomach-ka, leukopenia, allergic reactions. These drugs stimulate insulin secretion ??let?ami stop-dates of Langerhans. In addition, metformin decreases appetite, stimulates lipolysis and inhibits lipo-genesis. Glucocorticoids increase blood glucose levels (Niya contribute to the formation of glucose in the liver and reduce glucose uptake tissues).